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Recent advances have expanded the understanding of the complex pathophysiology of anemia of chronic kidney disease (CKD)
Multiple factors contribute to the pathophysiology of anemia of CKD, making it more than just an erythropoietin (EPO) and iron deficiency1
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One of these factors is the kidneys’ oxygen-sensing mechanism, which involves a hypoxia-inducible transcription factor called HIF1,2
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In CKD, the oxygen-sensing mechanism in the kidney is impaired, resulting in decreased EPO production. This contributes to the development of anemia1,2
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